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GSH is therefore a sensitive and reliable endogenous marker of oxidative stress

In addition, excessive NO produced by iNOS leads to cartilage injury by enhancing matrix metalloproteinase (MMP) activity and down regulating proteoglycan and collagen biosynthesis ( One study found that NO alone did not result in chondrocyte death but that NO triggered apoptosis when it reacted with O 2 - to produce chondrocytes (Nathan and Xie, 1994)

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