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Glutathione might inhibit the effects of cerebral infarction and enhance antiapoptotic signaling after ischemic stroke, suggesting that GSH may be a potent therapeutic antioxidant that can attenuate severe pathologies after ischemic stroke, and stimulating GSH synthesis through administration of GSH precursors and micronutrients like selenium can optimize GSH and GSH peroxidase for optimal antioxidant defense in cerebral ischemia (155, 156)

Its primary role is to protect cells from oxidative stress and damage caused by toxins and pollutants

A transgenic HER-2/neu mouse study reported changes in mammary-tumour development

However, in hyperthyroid individuals, levels of CD8-expressing cells are lower than in euthyroid individuals, indicating that prevalence of the Th2 response is associated with more pronounced symptoms (Bossowski et al ., 2003)