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His key contributions include: Discovery and initial characterization of MOTS-c as the first mitochondrial-encoded nuclear transcription regulator Demonstration of MOTS-c as an exercise-induced factor that improves age-dependent physical decline Elucidation of bi-genomic communication mechanisms between mitochondrial and nuclear genomes Investigation of MOTS-c polymorphisms associated with longevity in specific populations Studies on MOTS-c effects across aging, metabolic disease, and neurological applications His laboratory continues to investigate molecular mechanisms underlying MOTS-c function, therapeutic potential in age-related diseases, and translational applications for human health and longevity