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Unlike its influence on CAP-induced TRPV1 activation, exendin 939 did not block or potentiate proton-induced channel activity, even at a concentration approximately 30 times greater than its IC 50 value for inhibiting CAP-induced TRPV1 activity, indicating mode-specific inhibition of exendin 939

These findings highlight the potential importance of treatment persistence in maximizing the cardioprotective benefits of GLP-1RAs