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Adding further complexity, GLP-1RAs can stabilize the BBB (219, 220), raising the possibility that drugs with limited baseline uptake may improve their own CNS access over time, particularly in aging or NDD states where BBB dysfunction is common

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The co-expression of GLP-1R, glucokinase, and glucose transporter protein 2 (GLUT-2) in the neurons involved in the control of food intake suggests that these cells may play a role in glucose sensing in the brain [14, 16-19]