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Salicylic acid is keratolytic (it loosens dead skin) and anti-inflammatory, but inflamed skin is already a compromised barrier

The functional importance of the ASPL-VCP interaction in mediating VCP phosphorylation by ULK is further supported by our observation that the SG disassembly defects in cells expressing disease-causing VCP mutants or VCP bindingdefective ASPL mutants, both of which disrupt the ASPL-VCP interaction, were rescued by introducing phosphomimetic residues to bypass ULK-mediated phosphorylation

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doi: 10.1016/S0140-6736(16)31679-8 Erratum in: Lancet