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The activation of NF-B may lead to elevated androgen levels and disrupted glucose and lipid metabolism in PCOS patients.57 Inhibiting the NF-B-mediated inflammatory signaling pathway significantly improves the ovarian hormone microenvironment in PCOS rats.58 In line with these studies, the expression of TNF-, IL-6, and IL-1 NF-B increased in the ovaries of PCOS mice

Notably, BA exhibits dual cardiotoxic mechanisms: direct disruption of cardiomyocyte calcium dynamics, causing electrophysiological instability, or promotion of fibroblast-to-myofibroblast transition, which reduces cardiomyocyte membrane potential and slows conduction through gap junctions (Miragoli et al., 2011)

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doi: 10.1002/advs.202203707 52 CarabottiMSciroccoAMaselliMASeveriC