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This process may initiate the inappropriate incorporation of selenocysteine (SeCys) and selenomethionine (SeMet) into proteins, as well as impair their function, which may in turn lead to the development of oxidative stress, i.e., a state in which the oxidizing potential increases to the level that threatens the stability of cellular structures [14]

When these systems receive the nutrients they rely on, cellular metabolism can operate more efficiently and consistently

Protein aggregation in crowded environments

Environmental aggressors such as UV radiation, pollution, and dietary poisons may all enhance oxidative stress and cause skin damage