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Second, gluten-driven inflammation can impair epithelial tight junctions and amplify IL-15- and cytokine-rich mucosal stress responses, creating a milieu in which dysbiosis becomes self-reinforcing

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This is why its so important to understand the effects of this new class of drugs on it. In their review of the available medical literature, the researchers found that GLP-1 drugs improve certain measures of heart function, yet those improvements dont translate into significant improvements in VO 2 max