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These are neurotoxin induction-based models (1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP), 6-hydroxydopamine (6-OHDA) and agricultural pesticides (rotenone, paraquat)), pharmacological models (reserpine or haloperidol treated rats), and genetic models (-synuclein, leucine-rich repeat kinase 2 (LRRK2), DJ-1, phosphatase and tensin homolog (PTEN)-induced kinase 1 (PINK-1) and Parkin)

The rationale for studying them together is straightforward: if two compounds affect different but converging pathways involved in tissue repair, their combined application in experimental models may reveal interactions that cannot be observed when studying either compound in isolation

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View source Current Pharmaceutical Design 2016 PMID 27138887 Sikiric P et al