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Description

Neutrophil-derived 5-adenosine monophosphate promotes endothelial barrier function via CD73-mediated conversion to adenosine and endothelial A2B receptor activation

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The trial was designed as a non-inferiority trial with a pre-specified risk margin of 1.3 for the hazard ratio of MACE and was also powered for a superiority comparison if non-inferiority was demonstrated

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The Transitional Phase The transition off of a GLP-1 can serve as a "make or break" window for patients

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