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GLP-1RAs have been shown to reduce the activation of nuclear factor-kappa B (NF-B), a key regulator of inflammatory responses, thereby decreasing the production of pro-inflammatory cytokines such as TNF-, IL-6, and IL-1

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This possibly occurs by stimulation of the RANKL pathway and inhibition of production of the decoy receptor for RANKL/osteoprotegerin, which differs from findings from preclinical studies [92]