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Teo W-P Article: MULTIPLE SCLEROSIS AND RELATED DISORDERS

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Their mechanism in this context involves the downregulation of pro-inflammatory cytokines including interleukin-1 alpha and tumor necrosis factor that directly damage follicular epithelium and disrupt the dermal papilla's signaling function

The reduction of N-terminal pro-B-type natriuretic peptide (NT-proBNP) was 15% more than placebo, this being an important change in a surrogate marker for improving heart function