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Deficient tumor necrosis factor-related apoptosis-inducing ligand (TRAIL) death receptor transport to the cell surface in human colon cancer cells selected for resistance to TRAIL-induced apoptosis

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evaluated the plasma L-carnitine levels of subjects with high levels of TMAO ( n = 2,595) and found that the intake of large amounts of L-carnitine caused cardiovascular disease (CVD) and major adverse cardiac events (MI, stroke, or death)

The ablation of Atg5 suppresses autophagy and p62 degradation, which results in the persistent activation of nuclear factor (erythroid-derived 2)-like 2 (NFE2L2) by releasing the inhibition of KEAP1 on NFE2L2