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this increases oxidative stress and may be another factor contributing to ALD
Walker et al., 2010), mitochondrial associated membrane (MAM) dysfunction (Watanabe et al., 2016), ER-Golgi transport defects (Atkin et al., 2014
More plausibly, these agents may access the brain via regions lacking classical tight-junction architecture, such as the AP, subfornical organ, or median eminencecollectively known as circumventricular organs [67]
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