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Although both TNFR1 and TNFR2 can intermediate neuroprotective effects under specific conditions, most of the evidence indicates that TNFR1 signalling is primarily associated with neurodegenerative and pro-inflammatory outcomes, but TNFR2 signalling more consistently promotes neuroprotection and tissue repair

FIGURE 1 3.2.2 CSD theory Trigeminal afferent activation sets off the pain pathway, which sends information to the TG, the TNC, and finally to the brainstem and cortical structures involved in pain processing, resulting in pain (Zhang et al., 2011)

8 Bmal1 loss in adipocytes remodels circadian, metabolic

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