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Dimethyl fumarate induces coordinated DNA methylation changes in circulating leukocytes (including CD4+ T cells and monocytes), while interferon- produces targeted, cell-type-specific methylation shifts, demonstrating that MS-relevant epigenetic programs are reversible and may be corrected in tandem with the restoration of one-carbon flux (138,144-146)

Collectively, these findings indicated that P-Mn/TI@mHSA not only suppressed inflammatory cytokine expression but also serve as an effective regulator in promoting the phenotypic transition of macrophages from M1 to M2