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NNMT as a Therapeutic Target Evidence has shown that NNMT knockdown leads to an increase in energy expenditure in adipocytes [11] and a decrease in glucose output in hepatocytes [14]

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Theoretical and computational analysis is needed to understand how specific plasticity rules and calcium thresholds in interneurons should be regulated to reconcile associative learning with stability of neuronal operation, and the existence of corresponding mechanisms in diverse types of inhibitory neurons requires experimental validation

Mouse patatin-like phospholipase domain-containing 3 influences systemic lipid and glucose homeostasis