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FOXO4-DRI effectively counteracted doxorubicin-induced cell senescence and additionally mitigated doxorubicin-induced loss of body mass and elevated levels of aspartate aminotransferase (AST), contributing to the reduction of the hepatotoxicity of the chemotherapy agent [127]

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However, extracellular NAMPT induced through the TLR4 receptor activates signaling that triggers the differentiation and secretion of anti-inflammatory and tumorigenic cytokines and creating an immunosuppressive microenvironment

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