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Taken together, these data suggest that MG may contribute to ferroptosis by affecting GSH regeneration and its ability to reduce oxidative stress

These SM HLCs maintain a de-differentiated phenotype and for this reason, they are more appropriate for potentially replacing damaged hepatocytes and for supporting liver regeneration in vivo or for in vitro studies geared at understanding liver regeneration

Consequently, the inhibition of drug efflux transporters may cause the buildup of potentially harmful metabolites in hepatocytes, which can contribute to hepatotoxicity

Another key consideration is side effects, which can vary significantly from patient to patient