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Other studies have shown that selenocysteine in GPx significantly delays human amyloid- 2 -induced paralysis, which is positively correlated with the incidence of Alzheimers disease and recovers -amyloid-induced toxicity and reduces the cellular level of EOS, by positively affecting life span and age-related pathophysiological alterations [67, 68]

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PEG-MGF also raises product-specific unknowns, including whether the PEGylated form reaches the intended muscle tissue, how long it remains biologically active, and whether immune responses to PEG or the peptide occur [15]

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