In mouse models of highfat dietinduced liver steatosis, it significantly inhibits hepatic NNMT activity, reduces NAM methylation, increases NAD+ and SAM levels, enhances mitochondrial fatty acid oxidation, reduces hepatic triglyceride and lipid accumulation, lowers lipotoxicity markers (e.g., malondialdehyde, transaminases), improves hepatocellular injury, and reverses steatosis
We will then refer you for lab work to find out your baseline vitamin levels
The timeline for noticeable improvements can vary, as it depends on the underlying reason for the injection, the severity of the deficiency, and your individual health situation
The clinical trials through Metabolic Pharmaceuticals specifically documented this no adverse hormonal changes, no IGF-1 elevation, no glucose metabolism disruption