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Activation of this circuit in obese mice reduces food intake and improves glucose tolerance by stimulating insulin secretion via inhibition of GABA-input-dependent sympathetic nervous activity in the paraventricular hypothalamus

From 24.2% at 48 weeks to 28.7% at 68 weeks

The progressive aggregation of -syn into insoluble amyloid fibrils within neurons or oligodendrocytes, which leads to the formation of insoluble LBs and LNs under oxidative stress, is now recognized as a defining feature of PD and other synucleinopathies, including multiple system atrophy [47,48,49]

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