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In summary, despite that Rac1, Cdc42, and Rac1 are all involved in both insertion and internalization of AMPARs at the synapses, they appear to play distinct roles in LTP and LTD (Figure 2)

These findings underscore the urgent need for targeted antioxidant therapies capable of quenching this systemic ROS burden and restoring metabolic homeostasis

It demonstrated increased efficiency of oxidative phosphorylation in rat mitochondria in vitro and possible antioxidant effects are also attributed to it