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Thus, leptin-like insulin, represents adiposity signal inhibiting food intake by interacting with hypothalamic receptors activated through POMC and CART neuronal pathways stimulating satiety center, and reducing the activity of NPY/AgRP neurons driving the appetitive behavior

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Current pharmacological modulators targeting cGAS-STING signaling mainly include NiH, PEI-PDA@C-176 NPs, and FDL@TP, and these inhibitors have been confirmed to be effective in both in vitro experiments and murine arthritis (70, 71)

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