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Similarly, in CKD patients with transfusional iron overload, erythrocytes show increased osmotic fragility and higher eryptosis rates, correlating with serum non-transferrin-bound iron (NTBI) levels (Matarrese et al., 2005)

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The molecular gutliver signaling, we substantially revised the manuscript with detailed discussions of key pathways: TLR4/NF-B, MAPK signaling, tight junction regulation, microbial metabolite signaling (e.g., SCFAs), and cytokine cascades

MKC-231, a choline uptake enhancer, ameliorates working memory deficits and decreased hippocampal acetylcholine induced by ethylcholine aziridinium ion in mice