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Mechanistically, SSBP1 interacted with p53 and promoted DNA-PK-dependent phosphorylation of p53 at S15 to facilitate the nuclear accumulation of p53, which ultimately inhibited the transcription of SLC7A11 during ferroptosis in high fructose-treated glomerular podocytes (Fig

Inhibition of AP-1 and MAPK signaling and activation of Nrf2/ARE pathway by quercitrin