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First, its interplay with lipid metabolism: surplus pyruvate generated through glycolysis is converted to acetyl-CoA, which can enter lipid biosynthetic pathways and promote the synthesis of fatty acids, particularly PUFAs

273 Using the lipid overload oxidative stress-related calcification model, it was found that the downregulation of p53 expression suppressed GPX4 and SLC7A11 expression and decreased the GSH/GSSG ratio, thereby promoting lipid peroxidation and ferroptosis

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Angiogenesis, nitric oxide, endothelial signaling, vascular endothelial growth factor, and FAK-paxillin can all be research terms when tied to model-specific literature [4] [6] [9]