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This suggests a causal role of CD36 in steatosis supported by abnormally increased CD36 levels in NAFLD patients: a small study reported increased CD36 mRNA levels in obese subjects with high amounts of intracellular fat (66.0 6.8%, n = 5), compared to subjects with low liver fat content (6.4 2.7%, n = 5) [24]

Finally, to assess a functional indicator of oxidative damage and mitochondrial reactive oxygen species production, the activity of aconitase, a redox-sensitive enzyme in the tricarboxylic acid cycle, was measured

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