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10.1007/s00210-007-0216-0 387

This is mediated by the interaction of CD47 with the inhibitory macrophage receptor, SIRP (Signal regulatory protein alpha) [166]

The rationale is that under normal physiological circumstances, the decline in IGFBP-1 from its maximally stimulated overnight fasting level (34) to the level seen after food intake on a molar basis is relatively modest, and only leads to minor increases in serum free IGF-I (37, 38)

The significant IGF-1 increases may promote cellular adaptations and recovery processes, making it a compound of interest for growth and repair research