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In addition to the retention of mitochondrial Fe-S protein function under cystine starvation, we also find that protein expression is sustained during this time frame (Fig

Zhang W, Wang T, Xue, et al

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Conclusions In conclusion, our study elucidates the molecular responses of chondrocytes to H 2 O 2 -induced oxidative stress, highlighting the interconnected roles of ER stress, cytoskeletal remodeling, protein secretion, and immune response that are key factors associated with the progression of OA