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Across all these drugs, the common mechanism of action are glucose lowering via -cell GLP-1R, slowed gastric emptying (gut) and appetite suppression via central effects (McLean et al., 2021)

SCD patients have aberrant metabolic regulation of fumarate, glutamate, glycine, malate, and other metabolites involved in glycolysis, nucleotide catabolism, and glutathione metabolism 3,4,5,6

Given their critical roles in ferroptosis suppression, these antioxidant regulators represent promising therapeutic targets for counteracting ROS-mediated cytotoxicity (Mynott et al., 2023)

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