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Treatment with N-acetyl-L-cysteine, an antioxidant, or adenovirus-mediated overexpression of GPx1 attenuated the upregulation of adhesion molecules, confirming the role for GPx-1 suppression and hydrogen peroxide accumulation in these proinflammatory responses

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As increased GABA-ergic neurotransmission has been implicated in neuronal inhibition associated with HE, the efficacy of utilizing benzodiazepine receptor antagonists to treat HE has been evaluated in several RCTs

It can also cause nausea, vomiting, diarrhea, stomach pain, constipation, and/or anxiety and mood changes