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The findings of this work clearly indicate that ALC has a neurotherapeutic impact in a 6-OHDA-mediated model of PD by inhibiting apoptosis, neuroinflammation, astrogliosis, and OS, and that it might be considered as a potential supplementary treatment option for PD management (Afshin-Majd et al., 2017)

The significant 40% increase in MDA levels compared to fluconazole-treated or control cells suggests that CitAgNPs induce oxidative stress leading to lipid peroxidation in the fungal membranes [42]