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Whether liver-specific GSH synthesis was responsible for downregulated lipogenic gene expression and reduced serum triglycerides or whether these phenotypes were a byproduct of a liver-independent process in Gclc KO mice was unclear

Such method overcomes the expense and/or inconvenience of intravenous glutathione administration and also overcomes the increased likelihood of side-effects associated with the ingestion of high doses of glutathione

These findings suggest that the GCGR agonism of Retatrutide contributes to the reduction in circulating ANGPTL3/8, which in turn may lead to lower triglyceride and LDL-cholesterol levels

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