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Activation of the Nrf2ARE pathway in muscle and spinal cord during ALS-like pathology in mice expressing mutant SOD1

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AG induction was limited to 12 h and subsequent infections were only allowed to proceed for ~1 phage replication cycle (3035 min for T-phages and 6070 min for P1vir and vir) to prevent prolonged induction of various AGs from biasing the results

Remarkably, ALA treatment also prevented thickening and narrowing of the lumina of small arteries, the development of necrosis in the glomeruli and a mild hyperplasia of smooth muscles of an experimental model of hypertension (Vasdev et al., 2003)