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Fe 2 subsequently enters the cytosolic labile iron pool (LIP) through divalent metal transporter 1 (DMT1) and other non-transferrin-bound iron (NTBI) carriers [17]

While previous findings of increased total glutathione in subjects with asthma may reflect an adaptive response whereby synthesis and mobilization of GSH is increased to combat acute insults, these previous findings are more likely due to increased GSSG formation (206) since protein SHs are also reduced in these subjects (15, 236)

Reprogramming metabolism by histone methyltransferase NSD2 drives endocrine resistance via coordinated activation of pentose phosphate pathway enzymes