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Rather than stimulating hormone release, BPC-157 appears to work primarily through nitric oxide pathways, angiogenesis, and local tissue repair mechanisms

Reducing fibrosis and scarring, which is particularly beneficial for autoimmune diseases affecting the intestines and other tissues

For instance, depletion of mGSH results in Trx2 oxidation (Zhang et al., 2007), while hypercholesterolemic pigs with selective depletion of mGSH in heart mitochondria, exhibit decreased levels of the mitochondria-specific antioxidant enzymes such as SOD2, Trx2, and Prx3 (McCommis et al., 2011)

While both are starting doses designed for gastrointestinal tolerance building, they are not equivalent