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Clinical Endocrinology 73(5):637-643
albicans to albumin just prior to infection was sufficient to boost pathogenicity (Fig
In contrast, the pro-viral BHK-7 clone displayed a signature of immune evasion and developmental reprogramming: the Runt family of TFsknown for roles in immune suppression ( Figure 7A ), consistent with their ability to bind viral genomes ( MYC_N ), and developmental plasticity ( Homeobox, HLH ) were most prominent in infected BHK-7 cells ( Figures 7B, E )
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