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Chrysomycin a attenuates neuroinflammation by down-regulating NLRP3/Cleaved Caspase-1 Signaling Pathway in LPS-Stimulated Mice and BV2 cells

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Recent multi-omics analyses by Cappellani et al., for instance, have provided strong genetic and proteomic evidence linking DR pathogenesis directly to the activation of specific immune pathways, including the complement system and adaptive T-cell responses [53, 58, 59]

This sequence plays a role in glomerular injury, leading to albuminuria and eventual glomerulosclerosis