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The oxidation of NADPH provides electrons for biosynthesis

Its therapeutic value lies in its ability to selectively induce apoptosis in senescent cells by disrupting the FOXO4-p53 interactome, a mechanism that evades the collateral cytotoxicity often seen with broader senolytics like Navitoclax (Huang et al., 2021)

K., Chng, J

Constitutive activation of NRF2, mainly via loss-of-function mutations in the CUL3-adapter KEAP1 or gain-of-function mutations in NFE2L2 , is thought to provide a fitness advantage to tumor cells in confronting metabolic/oxidative stress resulting from dysregulated growth (DeNicola et al, 2011