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An increased proportion of PUFAs in normal diet also induces oxidative stress (increased malondialdehyde [MDA]), reduces cellular defense against oxidative stress (metallothionein) in the liver, and promotes oxidative degradation of DNA (elevated 8-hydroxy-2-deoxyguanosine levels serum) in a content-dependent manner despite increased fat oxidation and decreased fat synthesis [80]

[1] Those who cannot digest or absorb B12 as a result of inherited genetics or damage to the stomach and small intestine require more than can be absorbed from sublingual supplementation

Thats why GLP-1s show up in clinical trials for cardiovascular disease, kidney disease, heart failure, dementia, NASH (fatty liver), and cancer

Over time, this breaks down collagen, speeds up aging, and increases the risk of skin cancer