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Nussbaumer-StreitBGreenblattAKaminski-HartenthalerANoordMGVFornerisCAMorganLCet al

R.PaulS

The role of JNK as an amplification pathway for mitochondrial damage induced by APAP has been extensively investigated using different approaches, from pharmacological inhibition to antisense oligonucleotides-mediated knockdown of Jnk1 and Jnk2 or global Jnk2 deletion in mice (Han et al., 2013