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The complementary MOAs of amylin-analog and GLP-1 receptor agonists appear to be a promising combination therapy with greater weight loss than any other currently approved AOM and the potential to further close the gap with bariatric surgery outcomes

Nature 458:762765 Galan-Cobo A, Sitthideatphaiboon P, Qu X et al (2019) LKB1 and KEAP1/NRF2 Pathways cooperatively promote metabolic reprogramming with enhanced glutamine dependence in KRAS-mutant lung adenocarcinoma

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All of them work with the brain own repair mechanisms rather than against them