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Perhaps further research could uncover how properly addressing a malfunctioning ECS might prevent vulnerable people from entering psychotic states altogether

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Research suggests it may increase endogenous GLP-1 secretion from intestinal L-cells, providing a mild GLP-1 boosting effect that complements rather than replaces exogenous GLP-1 therapy

As a simple hypothetical example: if enhanced H 2 O 2 drives oxidation of 2 GSH to GSSG at 20 nmol.g 1 FW min 1 (which even if one considers only DHAR as a source of GSSG is no more than 1% of typical capacities measured in vitro ) but the in vivo GR activity is 1% slower (19.8 nmol.g 1 FW min 1