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8 Within neurologic contexts, glutathione is often discussed in relation to redox balance and oxidative stress, particularly in clinical presentations where inflammatory signaling or metabolic strain may be present

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NAA exerts its function in a histone acetyltransferase P300/CBP-associated factor (PCAF)-dependent manner, leading to lamin A acetylation, impairing its interaction with Sun2 (Sad1 and Unc84 domain-containing protein 2) and subsequent lytic granules, from CD8 + T cells and NK cells, polarization and release [214]