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353 Collectively, these genetic disorders underscore the non-redundant role of the UPS in preventing protein aggregation and preserving neuronal viability

Neurons, astrocytes, oligodendrocytes, microglia, endothelial cells, and pericytes all express GLP-1 receptors, positioning incretin signaling to influence neuronal metabolism, synaptic support, neuroinflammation, myelination, vascular integrity, and bloodbrain barrier function [22]

Most of the strongest evidence linking GLP-1 drugs to potential MS benefits comes from animal and laboratory studies