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The continued stimulation by Ox-LDL impacts the expression of cellular tight junction proteins (occludin) and the endothelial monolayer becomes hyperpermeable [109, 110], aggravating ECD
In contrast to these pharmacological effects in obesity, animal studies largely depict obesity-promoting effects of GIP and attribute the weight-regulating effects of GIP to CNS-GIPR signaling (14)
Thus, mitochondrial dysfunction has been identified in FXS and its animal models
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