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One study (87) provides evidence that pro-GLP-1 prevented apoptosis induced by oxygen-glucose deprivation (OGD) in cultured cortical neurons, and exerted neuroprotective effects through the cAMP/PKA and PI3K/Akt signaling pathways but not the ERK pathway in mice against cerebral ischemia
GLP-1 enhances glucose metabolism by stimulating insulin secretion when blood sugar levels rise, inhibiting glucagon release, and slowing gastric emptying to regulate nutrient absorption
Patients with certain GLP1R variants may reach effective appetite suppression at lower doses, while those with specific FTO variants may require longer adaptation periods between increases
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