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Soni A, Kabra SK, Lodha R

10.1093/oxfordjournals.bmb.a072641 92 RantforsJ.CassutoJ

108,187 Furthermore, the deletion of ZIP7 greatly enhances the ER stress response of proliferating progenitor cells, leading to apoptosis and disrupting intestinal epithelial cell proliferation and dryness

This review integrates mechanistic insights with translational advances, outlining how dysregulation of chaperones, autophagy-mitophagy, the ubiquitinproteasome system, and ER stress pathways drive human diseases, while highlighting emerging therapeutic platforms, from pharmacological chaperones and autophagy modulators to targeted protein degradation technologies, CRISPR screens, spatial biology, and AI-guided drug discovery